|
|
|
|
NTS encephalopathy was clinically characterized by diffuse and rapidly progressive brain dysfunction and circulatory failure that developed following enteritis. There was no evidence of severe dehydration or sepsis, and encephalopathy was r arely accompanied by abnormal laboratory data, except elevated CSF opening pressure, brain edema on CT, and slow waves on EEG. Pathologic findings included minimal ischemic damage and mild edema in the brain, microvesicular fatty change of the liver, se vere enterocolitis but no evidence of dehydration, and no fatal organ damage including microvasculature and endothelial cells. Noninfectious encephalopathy associated with nontyphoidal salmonella infection is a distinctive clinical entity that can be dif ferentiated from Reye's syndrome and Ekiri. |
|